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Etiologic types and stages of the major disorders of glucose tolerance are displayed.

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Master table with participants information and clinical variables in the format of SQLite database.

MethodTheoryReferenceAdsorptionDeposition of the enzyme on the electrode surface.

It involves the lysosomal degradation of cytoplasmic proteins or entire organelles for catabolic regeneration of nutrient pools.

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The authors stated that the mutation had a frequency of 100 in this population.

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Inconsistent and inaccurate on continuous tests with different strips.

National Institute of Diabetes and Digestive and .

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Kidney DiseasesDiabetes and prediabetes tests.

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Patients with hyperglycemia were subdivided between those with a previous history of diabetes and those without a previous history of diabetes.

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Using the EGP estimation method presented in these constrained hours significantly reduced BG fitting errors.

Although its effect on glycogenolysis rapidly wanes, hyperglycemia continues because the effects of epinephrine on gluconeogenesis and glucose disposal persist.

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Certain types of drugs including prescription drugs, over the counter drugs and herbal preparations may contribute to hyperglycemia.

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However, rather than being inhibited, the rate of pyruvate oxidation was increased approximately 300 in burned patients.

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We have established that the overall evidence for pharmacological neuroprotection is weak.

Carbohydrate containing foods directly affect your blood glucose level after eating, so reviewing the amount of carbohydrate in your meals and snacks may be helpful in determining the cause of hyperglycemia.

Moreover, hyperglycemia conferred resistance to chemotherapy for breast cancer but not nonmalignant cells, and this resistance was overcome by inhibiting fatty acid synthase or ceramide production.

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